Neuroreceptor Therapy
The Method of Proprioceptive-Deep Tendon Reflex (P-DTR) in Functional Neurology
The method of Proprioceptive Deep Tendon Reflex (P-DTR) was founded and developed by Dr. Jose Palomar and has been successfully used in clinical practice for several years. P-DTR is the first neurological manual therapy based on neurology, neurophysiology, biomechanics and basics of Applied Kinesiology. P-DTR is a neurological, reflexogenic system, which efficiently treats a wide spectrum of functional problems and solves musculoskeletal, gastrointestinal, hormonal, chemical and emotional dysfunctions. Dysfunction is a physiological and reflexory disorder of the internal organs, which in most cases has a compensatory character. The main goal of the P-DTR treatment is to restore optimal reflexive activity of the nervous system to stimulus. This includes its motor and gland response, which would result in no symptoms of pain or discomfort perceived by the client, optimal range of motion and accurate appropriate adaptation to the conditions of the external environment. In other words, Neurological Health restores this way.

P-DTR is a very gentle, non-invasive, and painless method based on the real time manual effect of particular sensory receptors on the human body. Doctor Palomar created a unique system of neurological challenges and has discovered the predictable “rules” that demonstrate how the CNS responds to stimulus. He has demonstrated extensively the difference in the CNS response under normal conditions (no dysfunction) and during the stimulation of a dysfunctional afferent input. The types of manual afferent inputs (stimuli) that are used can be produced in a variety of ways including light swiping (to stimulate the receptors of touch), local stretching (to stimulate Golgi receptors), deep pressure (Pacini receptors) and many more. Today, P-DTR works with most of the exteroreceptors, interoreceptors and proprioceptors that form the afferent input to the CNS.
The physiological explanation of this method is logical – each type of receptor (for example: Golgi, Pacini, vibration, nociceptors etc.) is stimulated and when the threshold for that receptor has been exceeded by the amount of stimuli, the stimuli are converted into electrical impulses. These electrical impulses form the afferent information that reaches the CNS with each type of input being relayed along their respective pathways. The CNS receives this information, interprets it and makes a motor or gland response based on the synthesis of all the information it has received. For example, the sensation of PAIN. It is synthesized directly in the brain and is a complex product of the information from the nociceptive, proprioceptive and exteroceptive systems. Put simply, the sensation of pain would be the interpretation of the brain based on a complex integration of information from variety of different sources.
The main principles of the P-DTR concept
• The CNS controls all functional processes of the body at physical, biochemical, endocrine, emotional and mental levels
• Any functional or dysfunctional changes are defined by CNS
• The CNS receives and interprets external and internal information 24/7
• Any motor, gland response or response of conscious thought will depend on the quality of received information and the quality of its interpretation
• Aberrant, distorted, excessive threshold signal from the receptors will cause neuromuscular, endocrine, or behavioral dysfunctions
• The main cause of dysfunctions is the aberrant afferent information coming in the CNS
• Neurological dysfunction can remain for an indefinite period of time affecting the optimal performance of the individual (in many cases long after physiological healing has been completed after a trauma)
• The CNS will constantly modulate incoming signals to be able to control the afferent flow, enabling the person to make sense of the world around them and respond appropriately
The P-DTR method provides the tools and assessments to locate and diagnose the dysfunctional receptors, to stimulate all parts of a dysfunction, to find the priority area, which is most important for the nervous system, to control afferent information flow and to reset it when it is dysfunctional, thereby altering the integration of all noxious stimuli and the formation of sense of pain itself.
The theoretical core of the method is pairing of dysfunctional afferent signals. The excessive afferent information, which arrives to CNS from the paired receptor fields and the quantitative change of the information flow from one field, inevitably leads directly to the change of the information flow from another field. In other words, any stimulus to the CNS from a dysfunctional receptor will be compensated.
A neurologically healthy and well-organized CNS constantly receives and analyses the incoming information to produce an appropriate motor and/ or gland response. In this way, we can say that when the threshold signals from receptors are within so-called “green zone” (optimal zone of normal function), this means that the information is under control, it is managed by the CNS and the CNS has enough resources for self-compensation, self-regulation and optimal daily performance. Those receptors are called “functional” and fire at a certain optimal range of action potential threshold.
A “dysfunctional” receptor has an altered action potential threshold (too high or too low), which means that it is extremely energy inefficient and itself is a source of systematic stress for CNS as this alteration to normal function must be compensated for.
In case of high signals from such receptors, the CNS will always choose to compensate for these signals, regardless of the consequential effects on the body, which can lead to a number of dysfunctions such as illnesses, instability, limited range of motion, lack of energy, emotional issues, etc.

Doctor Palomar studied the receptors with aberrant high signals and further discovered that only one of these dysfunctional receptors fields is always prioritized as the Primary dysfunctional zone. This receptor field has a high signal and is always compensated for by another receptor, which acts as the Main Secondary compensatory zone and also an excessive signal. If the compensation of MS is not enough, the brain creates other secondary virtual compensations, and if this is not sufficient, the brain will create new dysfunctions that act to increase the compensation for the Priority Primary receptor issue. Dr. Palomar then discovered that the dysfunctional high signal receptors could create fractals of compensations, the compensatory “tree”, which would affect the whole body and result in major physiological, endocrine and immune problems.
Doctor Palomar discovered that any dysfunctional signal would affect the myotatic reflex of the muscles (spindle cells) and therefore change the response of the muscles, as assessed using Manual Muscle Testing. The aberrant signal from any type of receptor field may cause the functional weakness or hypertonicity of a single muscle as well as it could potentially cause a global hypotonic or hypertonic response. All Primary, Secondary and Tertiary dysfunctional receptors have associated muscles and a particular inhibition pattern, depending on which level of the CNS the signal is blocked.
Once the dysfunctional receptor fields – both primary and secondary – are found and confirmed, the aberrant afferent flow can be “reset” by a specific stimulus to both receptor fields at the same time and elicitation of the Deep Tendon Reflex. Under simultaneous stimulation of the main dysfunctional receptor areas, the nervous system receives two signals, which compensate each other allowing the brain to “reset” the previous aberrant operational information.
Doctor Palomar discovered that DTR does not cause just a local reflex (as described in the textbooks), but affects the whole central nervous system and influences the neurological response of the brain. One of the possible explanations could be that as the deep tendon reflex itself is a corrective and/or damage control mechanism for the CNS, it allows the brain to evaluate afferent information and make an appropriate response immediately.
When stimulating both Primary and Secondary fields, the brain receives top priority information from those specific areas and becomes acutely sensitized to it. Eliciting a DTR helps the brain to reconstitute the aberrant information and reset it to normal levels. This means that the high signal level would decrease to a “green zone” level and there would be no need for the brain to compensate for it any longer. In majority of the cases, the result of this treatment is seen and felt immediately – pain symptoms disappear or decrease significantly, range of motion improves, associated muscles are tested as normotonic, and inhibition pattern disappears.

P-DTR works with different levels of neurological organization of the CNS. Dr. Palomar applied the same concept of aberrant receptor signals to the higher regulatory centers of the CNS and had successful results working with cranial nerves nuclei using abnormal breathing patterns to “reset” instead of DTR and other brain nuclei – e.g. basal ganglia, to regulate the motor system response.
The specific diagnostic, stimulation and treatment tools were developed for 12 pairs of cranial nerves (sensory, motor and visceromotor functions) and allow treating numerous neurological dysfunctions and symptoms. The MMT and specific stimulus to a particular nucleus are used to define a normal and abnormal response of the CNS in order to trace the primary dysfunction and the brain’s compensatory strategy for each particular cranial nerve dysfunction. Once the appropriate assessment is completed, the P-DTR protocol is used to confirm the context of the dynamics. When the Primary and Secondary dysfunction have been found, both dysfunctions are stimulated together with the appropriated abnormal breathing pattern being performed, to reset the CNS.
Currently, the P-DTR method is being researched and profoundly studied. The preliminary Electroneuromyographic research conducted by Department of Neuroscience, Physiotherapy and Reflex Therapy of Smolensk State Medical University clearly showed the interrelation between the primary and compensatory dysfunctional receptor fields and confirmed the hypothesis of pairing signals.
In order to analyze the state of central nociceptive and anti-nociceptive mechanisms, the method of exteroceptive suppression was used and showed the reduction of the inhibitory activity of interneurons of the cerebral-stem level providing nociceptive reactions, and this allows to assume that the P-DTR treatment affects not only the peripheral component of pain, but also the central anti-nociceptive structures which help to reduce the pain syndrome.
In order to analyze the state of skin vegetative reactions of a patient before and after the P-DTR treatment, the skin sympathetic potential was used before and after. In most cases, the sympathetic reaction of the patient’s skin decreased significantly.
Having observed a great number of patients treated by the method, we came to the conclusion that in some cases P-DTR can be applied as an independent treatment allowing patients to restore correct biomechanics of their bodies and solve many problems without pharmaceuticals. In other cases, P-DTR can be used as a supplementary treatment along with pharmaceuticals and treatment procedures of therapeutic and surgical nature. It is obvious that the treatment by P-DTR allows to restore correct proprioception, acquire normal tonicity of certain sets of muscles, improve posture, get rid of antalgic posture, align gait and further to raise the efficiency of therapeutic treatments such as a physical treatment and others.
Based on the principles taught in P-DTR, what José Palomar appears to do visually is more than a traditional postural assessment. Rather than looking for static “good vs. bad posture,” he seems to use posture as a window into which neurological compensation is currently dominant. A structured way to describe that process might be:
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Visual Neurological Survey
Step 1. Observe the patient’s resting organization
Before touching the patient, observe:
- Head position
- Eye level
- Mandible position
- Shoulder height and rotation
- Scapular resting position
- Thoracic rotation
- Rib cage asymmetry
- Pelvic tilt or rotation
- Weight distribution
- Knee position
- Foot progression angle
- Hand posture
- Finger resting position
- Muscle tone and visible guarding
The question is not:
“What is abnormal?”
The question is:
“What is the nervous system trying to protect?”
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Step 2. Look for a coherent pattern
A single asymmetry is usually not meaningful.
Instead, identify several findings that all suggest the same compensation.
Example:
- Right shoulder elevated
- Head translated left
- Left pelvis high
- Right foot externally rotated
These findings may represent one integrated neurological strategy rather than four separate musculoskeletal problems.
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Step 3. Estimate where the priority lies
The visual pattern often suggests which body region is likely driving the compensation.
Examples:
Upper-quarter priority
If you see:
- head tilt
- facial asymmetry
- jaw deviation
- eye asymmetry
- shoulder imbalance
you may suspect receptors involving:
- cranial structures
- TMJ
- cervical spine
- upper thorax
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Lower-quarter priority
If you see:
- pelvic rotation
- asymmetric weight bearing
- altered foot position
- femoral rotation
you may begin investigating:
- ankle
- knee
- hip
- pelvis
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Step 4. Look for protective behavior
Patients often protect an area before they report pain.
Examples include:
- never fully loading one leg
- holding one arm slightly abducted
- maintaining trunk rotation
- reduced arm swing
- avoiding cervical rotation
- persistent muscle guarding
These behaviors may indicate an active neurological adaptation.
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Step 5. Decide where to begin testing
The visual examination is not intended to make a diagnosis by itself.
Instead, it helps prioritize where manual testing is most likely to reveal an active dysfunction.
In other words:
Visual observation → hypothesis
Testing → confirmation
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Step 6. Continuously update the hypothesis
After each correction:
- posture changes
- breathing changes
- facial expression changes
- muscle tone changes
- weight distribution changes
These immediate changes help determine whether the treated dysfunction was part of the dominant compensation.
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Is it more than posture?
I would say yes.
What experienced practitioners often perceive is dynamic neurological organization, even while the patient is standing still. They are observing:
- preferred movement strategies
- resting muscle tone
- protective adaptations
- asymmetrical loading
- compensatory alignment
- how multiple body segments organize together
The posture itself is less important than the pattern it reveals.
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One additional point is worth emphasizing: much of this visual assessment relies on pattern recognition developed through extensive clinical experience. While practitioners can form hypotheses from observation, those hypotheses still need to be verified with reproducible examination findings (such as manual testing) before concluding that a particular area is the priority. Visual diagnosis guides the examination—it does not replace it.
